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Angiofilina (Amlodipine)
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Angiofilina

Angiofilina is used with or without other medications to treat high blood pressure. Lowering high blood pressure helps prevent strokes, heart attacks, and kidney problems. Angiofilina belongs to a class of drugs known as calcium channel blockers. It works by relaxing blood vessels so blood can flow more easily. Angiofilina is also used to prevent certain types of chest pain (angina).

Other names for this medication:
Abloom, Amlotens, Amlovasc, Ampin, Calbloc, Cardisan, Cardivas, Cordil, Cordipina, Dilotex, Diplor, Divask, Evangio, Goritel, Lodimax, Lodipine, Monovas, Nexotensil, Oralcam, Presovasc, Suplar, Tensigal, Tensocard, Vasocard, Vasonorm, Vilpin

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Also known as: Amlodipine

Description

{name} (amlodipine) is a calcium channel blocker that dilates (widens) blood vessels and improves blood flow.

{name} is used to treat chest pain (angina) and other conditions caused by coronary artery disease.

{name} is also used to treat high blood pressure (hypertension). Lowering blood pressure may lower your risk of a stroke or heart attack.

Amlodipine is for use in adults and children who are at least 6 years old.

Dosage

Take Angiofilina exactly as prescribed by your doctor. Follow all directions on your prescription label. Your doctor may occasionally change your dose. Do not use this medicine in larger or smaller amounts or for longer than recommended.

You may take Angiofilina with or without food. Take the medicine at the same time each day.

Your blood pressure will need to be checked often.

Your chest pain may become worse when you first start taking Angiofilina or when your dose is increased. Call your doctor if your chest pain is severe or ongoing.

If you are being treated for high blood pressure, keep using Angiofilina even if you feel well. High blood pressure often has no symptoms. You may need to use blood pressure medicine for the rest of your life.

Your hypertension or heart condition may be treated with a combination of drugs. Use all medications as directed by your doctor. Read the medication guide or patient instructions provided with each medication. Do not change your doses or stop taking any of your medications without your doctor's advice. This is especially important if you also take nitroglycerin.

Angiofilina is only part of a complete program of treatment that may also include diet, exercise, weight control, and other medications. Follow your diet, medication, and exercise routines very closely.

Store at room temperature away from moisture, heat, and light.

Detailed Angiofilina dosage information

Overdose

Seek emergency medical attention or call the Poison Help line at 1-800-222-1222.

Overdose symptoms may include rapid heartbeats, redness or warmth in your arms or legs, or fainting.

Storage

Store at room temperature between 15 and 30 degrees C (59 and 86 degrees F) away from moisture, light and heat. Keep container tightly closed. Throw away any unused medicine after the expiration date. Keep out of the reach of children in a container that small children cannot open.

Side effects

The most common side effects associated with Angiofilina are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.

Contraindications

Do not take Angiofilina if you are allergic to Angiofilina components. Do not take Angiofilina if you're pregnant or you plan to have a baby. Do not use it if you are a nursing mother. Do not use Angiofilina in case of suffering from significant aortic stenosis, cardiogenic shock, and unstable angina. Try to be careful with Angiofilina usage in case of having liver disease, heart failure or hepatic impairment. Do not use potassium supplements or salt substitutes. If you want to achieve most effective results without any side effects it is better to avoid alcohol. Be very careful when you are driving machine. Do not stop taking Angiofilina suddenly.

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A descriptive study identifying patients with SA from central Denmark, during the period 2006-2013, by the use of joint fluid culture data retrieved from the electronic database at the Department of Clinical Microbiology, Odense University Hospital. Additive effects of numbness and muscle aches on fatigue occurrence in individuals with HIV/AIDS who are taking antiretroviral therapy. NPX was significantly better than ERG in the overall efficacy of treatment rated by the patients (p less than 004). We confirm similar levels of shRNA singulair allergy medicine expression from the U6 promoter and the presence of processed/cleaved siRNAs for each construct in transduced MOLT-4 cells. Chemical parsing: Dissecting cell dependencies with a toolkit of selective BCL-2 family inhibitors. Furthermore, miR-181a increases the sensitivity of neuroblastoma cells to mitochondrial uncoupler-induced apoptosis, whereas miR-181a antagomir prevents cell death. Overall we suggest that a focus on the response process test takers go through will accelerate our understanding of faking behavior. To evaluate the effect of fibrates on fasting plasma triglycerides and other lipids levels in hypertriglyceridemia phenotypes with different genetic and clinical characteristics.

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Poor outcomes due to delayed diagnosis remain a problem worldwide. Characterization of CF4/CF3Br binary mixture adsorption on hydrophobic/hydrophilic surfaces via atomistic MD simulation. Five experimental studies investigated the process of choosing or generating such means for multiple goals. Methanol is an endogenous elicitor molecule for the synthesis of tryptophan and tryptophan-derived secondary metabolites upon senescence of detached rice leaves. In this paper, we focus on the disparities caused by viewpoint and scene changes and propose an efficient solution to these particular cases by adapting generic detectors, assuming xanax bars mg sizes boosting style. It is hoped that a prudent use or nonuse of these drugs might result in a reduction of thrombosis-associated diseases. The purpose of this article is to summarize recent developments in the understanding of NAFLD, essentially focusing on OxS as a major pathogenetic mechanism.

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